Full description
Near-infrared spectroscopy (NIRS)-derived skeletal muscle oxygenation (StO₂) remained preserved across five clinically accessible anatomical sites in stable chronic heart failure (HF) despite established microvascular abnormalities. Measurements were highly reproducible following sensor repositioning, supporting the reliability of NIRS for longitudinal monitoring. The inability of resting StO₂ to discriminate HF status suggests greater diagnostic value during physiological stress or acute HF decompensation.
Subjects
Cardiology (incl. cardiovascular diseases) |
Heart failure |
Near-infrared spectroscopy |
StO2 |
Tissue oxygenation |
microvascular function |
skeletal muscle oxygenation |
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Identifiers
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