MCO study tumour collection

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• Associated with A rapid PCR ELISA for the detection of activated K-ras in colorectal cancer
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• Associated with Activation of the K-ras oncogene in colorectal neoplasms is Associated with decreased apoptosis
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• Associated with Adverse Prognostic Effect of Methylation in Colorectal Cancer Is Reversed by Microsatellite Instability
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• Associated with CD103+ intraepithelial lymphocytes—a unique population in microsatellite unstable sporadic colorectal cancer
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• Associated with Characterisation and clinicopathological correlates of serum anti-p53 antibodies in breast and colon cancer
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• Associated with Clinicopathological significance of erbB-2 expression in colorectal carcinoma.
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• Associated with CpG island methylation in sporadic colorectal cancers and its relationship to microsatellite instability
  doi : 10.1053/GAST.2002.32997
  
• Associated with Detection of K-raspoint mutation by enriched PCR–colorimetric plate assay
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• Associated with Different APC genotypes in proximal and distal sporadic colorectal cancers suggest distinct WNT/β-catenin signalling thresholds for tumourigenesis
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• Associated with Epigenetic Inactivation of a Cluster of Genes Flanking MLH1 in Microsatellite-Unstable Colorectal Cancer
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• Associated with Factors Influencing the Detection of Mutant K-ras in the Serum of Patients with Colorectal Cancer
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• Associated with Germline epimutations of APC are not associated with inherited colorectal polyposis
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• Associated with Implementation of Novel Pyrosequencing Assays to Screen for Common Mutations of BRAF and KRAS in a Cohort of Sporadic Colorectal Cancers
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• Associated with Inactivation ofp16INK4a by CpG hypermethylation is not a frequent event in colorectal cancer
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• Associated with Kirsten ras mutations in patients with colorectal cancer: the ‘RASCAL II’ study
  doi : 10.1054/BJOC.2001.1964
  
• Associated with Large-scale delineation of secreted protein biomarkers overexpressed in cancer tissue and serum
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• Associated with MGMT methylation is associated primarily with the germline C>T SNP (rs16906252) in colorectal cancer and normal colonic mucosa
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• Associated with Methylation of the 3p22 region encompassing MLH1 is representative of the CpG island methylator phenotype in colorectal cancer
  doi : 10.1038/MODPATHOL.2010.212
  
• Associated with Microsatellite instability and the clinicopathological features of sporadic colorectal cancer
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• Associated with Microsatellite-stable diploid carcinoma: a biologically distinct and aggressive subset of sporadic colorectal cancer
  doi : 10.1054/BJOC.2000.1554
  
• Associated with Pathological and Genetic Correlates of Apoptosis in the Progression of Colorectal Neoplasia
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• Associated with RAD21 cohesin overexpression is a prognostic and predictive marker exacerbating poor prognosis in KRAS mutant colorectal carcinomas
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• Associated with Relative Distribution of Folate Species Is Associated with Global DNA Methylation in Human Colorectal Mucosa
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• Associated with Routine testing for mismatch repair deficiency in sporadic colorectal cancer is justified
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• Associated with Sporadic Colorectal Cancers With Microsatellite Instability and Their Possible Origin in Hyperplastic Polyps and Serrated Adenomas
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• Associated with Survival in stage II/III colorectal cancer is independently predicted by chromosomal and microsatellite instability, but not by specific driver mutations
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• Associated with The CpG Island Methylator Phenotype Is Not Associated with a Personal or Family History of Cancer
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• Associated with The Relationship between Hypomethylation and CpG Island Methylation in Colorectal Neoplasia
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• Associated with The Role of hMLH1 Methylation in the Development of Synchronous Sporadic Colorectal Carcinomas
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• Associated with The collagenase-1 (MMP-1) gene promoter polymorphism - 1607/2G is associated with favourable prognosis in patients with colorectal cancer
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• Associated with The role of MYH and microsatellite instability in the development of sporadic colorectal cancer
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• Associated with Tolerance of Whole-Genome Doubling Propagates Chromosomal Instability and Accelerates Cancer Genome Evolution
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• Associated with pubmed.ncbi.nlm.nih.gov/9196805/
  uri : pubmed.ncbi.nlm.nih.gov/9196805/
  
• Associated with ar.iiarjournals.org/cgi/content/long/32/1/73
  uri : ar.iiarjournals.org/cgi/content/long/32/1/73
  
• Associated with www.jimmunol.org/content/163/4/2276.abstract
  uri : www.jimmunol.org/content/163/4/2276.abstract
  
• Associated with cancerres.aacrjournals.org/content/62/1/53.abstract
  uri : cancerres.aacrjournals.org/content/62/1/53.abstract
  
• Associated with clincancerres.aacrjournals.org/content/9/7/2642.long
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