grant

Functions of viral chemokine receptor homologues important for cytomegalovirus pathogenesis and latency [ 2010 - 2013 ]

Also known as: Herpesvirus proteins that modify activities of the cell to promote virus infection and persistence. How do they work ?

Funded by National Health and Medical Research Council
Managed by The University of Queensland
Managed by University of Queensland
Provided by   National Health and Medical Research Council

Research Grant

[Cite as https://purl.org/au-research/grants/nhmrc/631401]

Researchers: A/Pr Nicholas Davis-Poynter (Principal investigator) ,  Dr Helen Farrell

Brief description Cytomegalovirus (CMV) causes life-threatening disease in babies, transplant recipients and HIV-AIDS patients. We will focus on a CMV gene that has been 'hijacked' from the host cell and enables the virus to switch on signalling molecules within infected cells. We will determine how these signals enable CMV to infect sites of the body that are critical for virus transmission and contribute to long-term virus persistence. Our results will provide new strategies for drugs against CMV.

Funding Amount $AUD 461,597.97

Funding Scheme NHMRC Project Grants

Notes Standard Project Grant

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Subjects
Medical and Health Sciences | Medical Microbiology | Medical Virology | cytomegalovirus | host-pathogen interaction | infectious disease | microbiology-virology | understanding virus-host interactions | viral chemokine receptor homologue | viral persistence | virus persistence and latency |
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ACN 633 798 857